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6%) had abnormal PCI. HVs and patients with different esophageal motility disorders had significantly different PCI (P less then .01). Type 1 achalasia patients had weaker PCI than patients with absent contractility (P = .02). Patients with abnormal PCI had more severe dysphagia (P = .02), nausea (P = .03), vomiting (P = .03), and lower bolus clearance (P less then .01) than patients with normal PCI. Conclusions and inferences Abnormal PCI was found in a fourth of patients with esophageal symptoms. PCI may be useful to distinguish some e
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