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Moreover, NAOs markedly blocked histopathological changes and collagen accumulation, as shown by HE and Sirius red staining, respectively. Finally, NAOs antagonized the CCl4-induced upregulation of the protein and mRNA levels of fibrogenic genes in the liver. In conclusion, our findings suggest that NAOs may be a promising candidate for the prevention and treatment of chronic liver injury via inhibition of the TGF-β/Smad signaling pathway.The synthesis, characterization and photoreduction ability of a new class of carbon dots made fr
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