https://mad2signals.com/index.....php/alterations-in-r
We disclosed that CaMKII had been built-in to the procedure through which Zn2+ facilitated the release of glutamate. Furthermore, a glutamate transporter (EAAT) was the molecular entity when it comes to action of Zn2+ on glutamate uptake through which Zn2+ reduces glutamate supply. Taken collectively, we show a novel action of Zn2+ , which can be to biphasically regulate glutamate homeostasis via Zn2+ concentration-dependent synaptic facilitation and depression. Hence, co-
Like
Comment
Share
