https://www.selleckchem.com/products/BIBW2992.html
Objective To investigate the underlying molecular mechanisms of brain injury in rats after cardiac arrest and cardiopulmonary resuscitation (CPR) by observing necroptosis of brain cells and changes of 90 cytokines in brain tissue. Methods Sprague-Dawley (SD) rats were divided into Sham group (n = 1 and cardiac arrest group (n = 1 according to random number table method. The model of asphyxia cardiac arrest for 6 minutes followed by CPR model was established. Tracheal intubation in Sham rats were routinely performed without inducing
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