https://www.selleckchem.com/products/gdc-0068.html
NAMPT was a target of miR-149-5p, miR-149-5p attenuated IL-1β-induced growth arrest and ECM degradation in chondrocytes, which was abolished by NAMPT overexpression. Importantly, circ_0008956 served as a sponge for miR-149-5p to up-regulate NAMPT expression in chondrocytes. Circ_0008956 contributed to IL-1β-induced growth arrest and ECM degradation in chondrocytes via miR-149-5p/NAMPT axis, suggesting a new insight into the pathogenesis of OA and a promising therapeutic target for OA treatment. Intravenous immunoglobulins (IVIg) have b
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