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Besides, the expression of signaling pathway was validated by signaling inhibitor. In comparison to normal cells, LUCAT1 was highly expressed in human pancreatic cancer cell lines (p0.05). The higher expression of LUCAT1 resulted in enhanced pathogenesis of PDA cells and motivated the development to S phase by regulation of cyclin D1, CDK4. Furthermore, LUCAT1 promoted PDA cells development by inducing AKT's and p38 MAPK's phosphorylation. LUCAT1, as the key factor, played a positive role in the proliferation and invasion of pancreat
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