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centrations were greater (P less then 0.05) in MI-IUGR lambs, plasma tumor necrosis factor α (TNFα) was reduced (P less then 0.05). FDS muscle contained greater (P less then 0.05) TNF receptor 1 (TNFR1) and IκBα protein content. These findings indicate that maternofetal inflammation in late pregnancy results in fetal programming that impairs growth capacity, muscle glucose oxidation, and lipid homeostasis in offspring. Inflammatory indicators measured in this study appear to reflect heightened cytokine sensitivity in muscle and compensat
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