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Therefore, in this review, we focused on some new pathogenetic elements involved in graft fibrosis (including epithelial/endothelial to mesenchymal transition, oxidative stress, activation of Wnt and Hedgehog signaling pathways, fatty acids oxidation and cellular senescence) that, in our opinion, could become in future good candidates as potential biomarkers and therapeutic targets.Chapter 12 was inadvertently published with the contributing authors listed as Mihaela Badea, Akhtar Hayat, and Jean-Louis Marty, whereas it should have been