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Our results support a model in which the isoform-specific N-terminal extensions affect chemomechanical coupling by combined steric and allosteric effects, thereby reducing both the length of the working stroke and the rate of ADP release in the absence of external loads by a factor of two for myosin-1C35 As the large change in maximum power output shows, the functional differences between the isoforms are further amplified by the presence of external loads. Little is known about what hospital and emergency department (ED) factors predict
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