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Heightened mitochondrial task results in increasingly increasing hypoxia from the renal cortex to the renal medulla. Renal hypoxia is prominent in hypertensive rats due to increased salt reabsorption inside the nephrons, which requires greater energy production by oxidative phosphorylation (OXPHOS). Consequently, spontaneously hypertensive rats (SHR) display better air deficiency (hypoxia) than normotensive Wistar Kyoto rats (WKY). Here, we desired to research the appearance of crucial proteins for
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