https://www.selleckchem.com/pr....oducts/BMS-536924.ht
Our results indicated that EGCG could recover the activity of autophagy in the TGF-β1-treated cells. Moreover, treatments with EGCG significantly decreased the α-SMA expression. Taken together, these findings revealed that autophagic regulation involved in the action of EGCG against TGF-β1-induced transformation of Tenon's fibroblasts. Through increasing intracellular autophagy, EGCG could be a potential anti-fibrotic reagent for preventing subconjunctival fibrosis after glaucoma filtration surgery. Pyroptosis and inflammation are in
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