https://www.selleckchem.com/products/SGI-1776.html
Background Several studies have proved that physical activity (PA) regulates energetic metabolism associated with mitochondrial dynamics through AMPK activation in healthy subjects. Obesity, a condition that induces oxidative stress, mitochondrial dysfunction, and low AMPK activity leads to mitochondrial fragmentation. However, few studies describe the effect of PA on mitochondrial dynamics regulation in obesity. Aim The present study aimed to evaluate the effect of a single session of PA on mitochondrial dynamics regulation as well as