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The clinical phenotype of the rare behavioural variant of Alzheimer's disease (bvAD) is insufficiently understood. Given the strong clinico-anatomical correlations of tau pathology in AD, we investigated the distribution of tau deposits in bvAD, in-vivo and ex-vivo, using positron emission tomography (PET) and postmortem examination. For the tau PET study, seven amyloid-β positive bvAD patients underwent [ F]flortaucipir or [ F]RO948 PET. We converted tau PET uptake values into standardised (W-)scores, adjusting for age, sex and mini m