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Heart failure with preserved ejection fraction (HFpEF) is an increasingly prevalent disease. Physical exercise has been shown to alter disease progression in HFpEF. We examined cardiomyocyte Ca homeostasis and left ventricular function in a metabolic HFpEF model in sedentary and trained rats following 8weeks of moderate-intensity continuous training (MICT) or high-intensity interval training (HIIT). Left ventricular in vivo function (echocardiography) and cardiomyocyte Ca transients (CaTs) (Fluo-4, confocal) were compared in ZSF-1 obese
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