https://www.selleckchem.com/products/Nolvadex.html
nder physiological and pathophysiological conditions become better defined, the current study may inform animal model selection for preclinical studies. Pulmonary arterial hypertension (PAH) is a complex disease of the small pulmonary arteries that is mainly characterized by vascular remodeling. It has been demonstrated that excessive proliferation of pulmonary arterial smooth muscle cells (PASMCs) plays a pivotal role in vascular remodeling during PAH. The present study was undertaken to explore the role of TMEM16A in regulating PASMC
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