https://www.selleckchem.com/pr....oducts/ginsenoside-r
Previous studies have shown that lncRNA NEAT1 and miR-29b are closely associated with repair of the injured spinal cord. However, the mechanism by which lncRNA NEAT1 promotes regeneration after spinal cord injury by regulating miR-29b has not been reported. To explore this mechanism, we established a rat model of spinal cord injury (SCI). The experimental rats were randomly assigned to one of six groups the sham, model, si-NEAT1, miR-29b, si-NEAT1 + negative control and si-NEAT1 + si-miR-29b groups. The hind limb motor function
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