https://www.selleckchem.com/btk.html
, arginine and proline metabolism, beta-alanine metabolism, citrate cycle (TCA cycle), glycerophospholipid metabolism and linoleic acid metabolism. 2MW, 3MW and 4MW could invert the pathological process of hyperuricemia to varying degrees through in part regulating the perturbed lipid metabolic pathway. 4MW were better than 2MW and 3MW in the intervention of the disordered tricarboxylic acid metabolism and purine metabolism caused by hyperuricemia. In summary, ECFs treatment could effectively alleviate symptoms of hyperuricemia and regulate metaboli