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Experiments in Xenopus oocytes and flies indicate that Hodor is a pH-sensitive, zinc-gated chloride channel that mediates a previously unrecognized nutritional preference for zinc. Hodor manages systemic growth from a subset of enterocytes-interstitial cells-by promoting food intake and insulin/IGF signalling. Although Hodor sustains gut luminal acidity and restrains microbial loads, its effect on systemic growth results through the modulation of Tor signalling and lysosomal homeostasis within interstitial cells. Hodor-like ge
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