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Using adenoviral-mediated TMEM16A increase in healthier primary peoples PAECs in vitro and in personal pulmonary arteries ex vivo, we illustrate the practical consequences of the augmented TMEM16A activity alterations of Ca2+ dynamics and eNOS activity as well as diminished NO production, PAECs proliferation, wound healing, tube formation and acetylcholine-mediated relaxation of real human pulmonary arteries. We suggest that th
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