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Pathogens and humans share an intrinsic relation related to molecular mimicry in their antigens. Interactions between immune system and pathogenic antigens result in a production of antibodies that could protect against infection, but develop autoreactive responses mediated by autoantibodies that react to pathogenic and human antigens because they share epitopes. In this study, a pipeline of bioinformatic tools was used to explore the repertory of autoantigens implicated in the develop of Systemic Lupus Erythematosus and their homologou