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Likewise, PKCα positively modulated TSHr expressions to strengthen the TSH/TSHr signal in the HPT axis, thereby synergistically contributing to TH biosynthesis. Moreover, β-CYP also disturbed TH biotransformation and biotransport by inducing DIO1 and inhibiting DIO3 in thyroids and TTR expressions in livers. Taken together, β-CYP has the thyroid-disturbing effect and could promote TH biosynthesis, and PKCα plays vital roles in β-CYP-caused hyperthyroidism.This global systematic analysis and review investigate the impacts of previous lan

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