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Diabetic persistent cutaneous ulcers (DCU) is one of the serious complications of diabetes mellitus, which mainly occurs in diabetics with peripheral neuropathy. Present studies have indicated that microRNAs (miRNAs/miRs) and their particular target genes are crucial regulators of cellular physiology and pathology including biological procedures which can be mixed up in legislation of diabetes and diabetes-related microvascular problems. In vivo and in vitro models have r

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ain effective self-cleaning, anti-fouling or heat-transfer abilities in harsh operating environments.Mitochondria take up Ca2+ through the mitochondrial calcium uniporter complex to regulate energy production, cytosolic Ca2+ signalling and cell death1,2. In mammals, the uniporter complex (uniplex) contains four core components the pore-forming MCU protein, the gatekeepers MICU1 and MICU2, and an auxiliary subunit, EMRE, essential for Ca2+ transport3-8. To prevent detrimental Ca2+ overload, the activity of MCU must be tightly regulated b